By Anil K. Mandal M.B.B.S., F.A.C.P. (auth.)
Anil okay. Mandai, M.D., is without doubt one of the trailblazers within the use of the transmission electron microscope within the examine of the urinary sediment. during this ebook, he studies his vast efforts to tie his big scientific expe rience to his stylish easy learn with the electron microscope. the photographs are accomplished, and the scientific correlates are well defined in tables and textual content. it might probably astonish a few readers ebook for fellows and scientific nephrol ogists has been written at the use of the transmission electron microscope within the examine of urine. a few may well view this as a worldly learn software. I, in spite of the fact that, applaud the trouble. such a lot of discoveries and advances in easy technological know-how lie unutilized simply because clinicians aren't conscious of the instruments to be had or have little guide of their use. possibly that's the reason why such a lot of assessments have come and long gone, were came across lifeless and dropped, or have easily been deserted after being judged too complicated-some simply because they have been, others simply because they have been by no means utilized and interpreted thoroughly. the total box of analysis appears to be like pulling forward and clear of scientific medication. accordingly, an attempt like this one, which speedily and obviously attempts to introduce a sophisticated learn exam method into medical drugs, is priceless of admiration and sup port.
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Extra resources for Assessment of Urinary Sediment by Electron Microscopy: Applications in Renal Disease
Most of the mitochondria (M) are swollen and devoid of cristae. though a few (between arrows) appear intact (UA + Le. 000). (Fig. 3-5) and abundant lysosomes (Fig. 3-6), the possibility of a reversible process remained. Because of the results of the urinary sediment study, the prospect of a good recovery of renal function was considered unlikely for this patient. 2, H. , a 61-year-old white male, was admitted to the Veterans Administration Medical Center, Augusta, Georgia on February 15, 1983, for the repair ACUTE RENAL FAILURE 47 Figure 3-4.
3. Naccarato WF, Caffo AL: The performance characteristics of the urinary protein method for the Du Pont ACA Analyzer. Du Pont Clinical Systems Technical Service Bulletin 1982. 4. Springberg PO, Garrett LE, Thompson AL, et al: Fixed and reproducible orthostatic proteinuria: Results of a 20 year study. Ann Intern Med 1982; 97:516--519. 5. Ganeval 0, Noel LH, Preud'Homme JL, et al: Light chain deposition disease: Its relation with AL-type amyloidosis. Kidney Int 1984; 26:1-9. 6. Alpers CE, Hopper J, Biava CG: Light chain glomerulopathy with amyloid-like deposits.
Functional Proteinuria In functional proteinuria, there are no demonstrable histopathological changes in the glomeruli and tubules. Urinary protein excretion increases two- to threefold following heavy exercise, febrile illness, congestive cardiac failure, emotional stress, norepinephrine infusion, and the prolonged assumption of the lordotic position; there is also increased formation of hyaline casts. This increase in proteinuria is attributable to a decrease in renal blood flow, with glomerular ischemia, and an increase in glomerular permeability to albumin.
Assessment of Urinary Sediment by Electron Microscopy: Applications in Renal Disease by Anil K. Mandal M.B.B.S., F.A.C.P. (auth.)