By Stephen M. Bonsib (auth.)
The kidney is an organ with advanced organogenesis vulnerable to quite a few misadventures in improvement and is uncovered to a various array of insults of hematogenous and reduce urinary tract foundation. This Atlas of scientific Renal Pathology presents an outline of the improvement, macroscopic and microscopic beneficial properties of the traditional kidney. this can be via a entire survey of developmental and cystic kidney ailments, vascular illnesses and tubulointerstitial ailments. An emphasis is put on gross diagnostic findings with precise histological correlates. moreover, the histological, immunofluorescent, immunohistochemical and ultrastructural good points of the key glomerular ailments and renal transplantation pathology are awarded. This compendium of non-neoplastic kidney ailments illustrates the overwhelming majority illnesses you will definitely come upon in surgical and post-mortem pathology.
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43 ARPKD, neonatal onset. The liver in neonatal ARPKD has a characteristic bile duct plate abnormality. The bile ducts reside in an expanded portal triad and are peripherally arrayed and branched. This liver abnormality may be a useful finding in cases in which the renal lesion is less severe and the diagnosis is more challenging. However, several other cystic kidney diseases also may have a bile duct plate malformation and develop congenital hepatic fibrosis Fig. 44 Liver in ARPKD, childhood onset.
Notice that the ureters are thickened and the pelves appear dilated, consistent with the obstructive complication of this anomaly (From Zhou M, Magi-Galluzzi C, editors. Genitourinary pathology. Philadelphia: Elsevier; 2007. p. 241; with permission) 19 dysplasia. This section presents a modest sampling of the many possible defects and combination of defects. 1 Rotation Anomaly Rotation anomalies are recognized by the abnormal location of the renal pelvis. Incomplete rotation results in an anterior location.
This developmental abnormality strengthens the postulate that segmental hypoplasia may have a developmental basis, at least in some cases Fig. 21 Segmental hypoplasia. This is the hypoplastic focus from the specimen in Fig. 20. Notice the abrupt transition from normal cortex to the hypoplastic focus. In this lesion, stroma predominates, with a few tubules and several glomeruli. No atrophic tubules or sclerotic glomeruli are present to indicate an atrophic lesion Fig. 23 Segmental hypoplasia. 5-cm kidney.
Atlas of Medical Renal Pathology by Stephen M. Bonsib (auth.)